The Resource Cerebral venous system in acute and chronic brain injuries, Min Lou, Jianmin Zhang, Yilong Wang, Yan Qu, Wuwei Feng, Xunming Ji, John H. Zhang, editors
Cerebral venous system in acute and chronic brain injuries, Min Lou, Jianmin Zhang, Yilong Wang, Yan Qu, Wuwei Feng, Xunming Ji, John H. Zhang, editors
Resource Information
The item Cerebral venous system in acute and chronic brain injuries, Min Lou, Jianmin Zhang, Yilong Wang, Yan Qu, Wuwei Feng, Xunming Ji, John H. Zhang, editors represents a specific, individual, material embodiment of a distinct intellectual or artistic creation found in Sydney Jones Library, University of Liverpool.This item is available to borrow from 1 library branch.
Resource Information
The item Cerebral venous system in acute and chronic brain injuries, Min Lou, Jianmin Zhang, Yilong Wang, Yan Qu, Wuwei Feng, Xunming Ji, John H. Zhang, editors represents a specific, individual, material embodiment of a distinct intellectual or artistic creation found in Sydney Jones Library, University of Liverpool.
This item is available to borrow from 1 library branch.
- Language
- eng
- Extent
- 1 online resource (249 pages).
- Note
- Hyperintense Signals in Veins or Sinus
- Contents
-
- Intro; Contents; Chapter 1: Neurovascular Network as Future Therapeutic Targets; 1 Introduction; 2 The Mismatch Between Preclinical Models and Clinical Types; 2.1 Recanalization Leads to a Small Infarction; 2.2 Unsuccessful Recanalization Leads to a Small Infarction; 2.3 Recanalization Leads to Bleeding, Edema, and Massive Infarction; 2.4 Vein Compression Involved in Reperfusion Injury; 2.5 Infarction Is Reversible Even After Days; 3 Establishment of New Stroke Pathophysiology to Address Clinical Issues; 3.1 Emphasis on Recirculation: Both Arterial and Venous Blood Flow
- Basic Rules for Recirculation (Fig. 1.7)Anatomy of the Cerebral Venous System; Venous Flow During Ischemic Stroke; Venous Flow During Intracranial Hemorrhage; Venous Flow During Subarachnoid Hemorrhage; Venous Flow During Traumatic Brain Injury; Recirculation as an Emerging Understanding of Stroke and Other Acute Brain Injuries; 3.2 Define Venous Function after Acute Arterial Stroke; Situations to Evaluate Venous Function; Animal Models and Technologies for Studying Venous Function; 3.3 Proposed Concept of Collateral Homeostasis; Hierarchy of Cerebral Collaterals
- Hemostasis in Both Arterial and Venous Collaterals (Fig. 1.11)Multimodal Monitoring Technologies for Collaterals; 4 Neurovascular Networks as Future Therapeutic Targets; 4.1 Pericytes as a Potential Interventional Target; 4.2 Smooth Muscle Phenotype for Autoregulation; 4.3 Collaterals for Recirculation; 5 Conclusion; References; Chapter 2: Animal Models of Venous Stroke; 1 Selection of Animals for Venous Stroke; 2 Early Cerebral Venous Thrombosis Models; 3 Cerebral Sinus and Venous Thrombosis (CSVT) Model; 3.1 Protocol (for Rat) [5, 14-17]; 3.2 Outcome Evaluation; Physiological Measures
- Local Cerebral Blood Flow (LCBF) MeasurementHistology; 4 Photochemical Dye Induced Cortical Vein Occlusion Model; 4.1 Protocol (for Rat) [20-22, 26]; 4.2 Outcome Evaluation; Local Cerebral Blood Flow Measurement; Histology; 5 Ferric Chloride Induced Superior Sagittal Sinus Thrombosis; 5.1 Protocol; Rat [28, 29]; Mouse [2, 3]; 5.2 Outcome Evaluation; Magnetic Resonance Imaging; Macroscopy, Micro- and Nano-Computed Tomography; References; Chapter 3: Imaging of Cerebral Vein in Acute Brain Injury; 1 Traumatic Brain Injury; 1.1 Computed Tomography (CT); Air in Veins
- 1.2 Susceptibility Weighted Imaging (SWI)Deep Cerebral Veins Asymmetry; Multifocal Signal Loss at Bridging Veins; 1.3 Digital Subtracted Angiography (DSA); Early Venous Filling; 1.4 Summary; 2 Cerebral Venous Thrombosis (CVT); 2.1 Computed Tomography; Dense Triangle Sign and Cord Sign; Focal Distension of the Venous Compartment; 2.2 CT Venography; Empty Delta Sign; 2.3 Magnetic Resonance Imaging; Regular MRI; T1-Weighted Isointense and T2-Weighted Hypointense; Signal Void or Blooming Artifact; High Signal in Sinus; Magnetic Resonance Venography (MRV); Filling Defect in Veins or Sinus
- Isbn
- 9783319960524
- Label
- Cerebral venous system in acute and chronic brain injuries
- Title
- Cerebral venous system in acute and chronic brain injuries
- Statement of responsibility
- Min Lou, Jianmin Zhang, Yilong Wang, Yan Qu, Wuwei Feng, Xunming Ji, John H. Zhang, editors
- Language
- eng
- Cataloging source
- EBLCP
- Dewey number
- 617.4/81044
- Index
- no index present
- LC call number
- RD594
- Literary form
- non fiction
- Nature of contents
- dictionaries
- http://library.link/vocab/relatedWorkOrContributorName
-
- Lou, Min
- Zhang, Jianmin
- Wang, Yilong
- Qu, Yan
- Feng, Wuwei
- Ji, Xunming
- Zhang, J. H.
- Series statement
- Springer Series in Translational Stroke Research
- http://library.link/vocab/subjectName
-
- Brain
- Cerebral veins
- Label
- Cerebral venous system in acute and chronic brain injuries, Min Lou, Jianmin Zhang, Yilong Wang, Yan Qu, Wuwei Feng, Xunming Ji, John H. Zhang, editors
- Note
- Hyperintense Signals in Veins or Sinus
- Antecedent source
- file reproduced from an electronic resource
- Carrier category
- online resource
- Carrier category code
-
- cr
- Carrier MARC source
- rdacarrier
- Content category
- text
- Content type code
-
- txt
- Content type MARC source
- rdacontent
- Contents
-
- Intro; Contents; Chapter 1: Neurovascular Network as Future Therapeutic Targets; 1 Introduction; 2 The Mismatch Between Preclinical Models and Clinical Types; 2.1 Recanalization Leads to a Small Infarction; 2.2 Unsuccessful Recanalization Leads to a Small Infarction; 2.3 Recanalization Leads to Bleeding, Edema, and Massive Infarction; 2.4 Vein Compression Involved in Reperfusion Injury; 2.5 Infarction Is Reversible Even After Days; 3 Establishment of New Stroke Pathophysiology to Address Clinical Issues; 3.1 Emphasis on Recirculation: Both Arterial and Venous Blood Flow
- Basic Rules for Recirculation (Fig. 1.7)Anatomy of the Cerebral Venous System; Venous Flow During Ischemic Stroke; Venous Flow During Intracranial Hemorrhage; Venous Flow During Subarachnoid Hemorrhage; Venous Flow During Traumatic Brain Injury; Recirculation as an Emerging Understanding of Stroke and Other Acute Brain Injuries; 3.2 Define Venous Function after Acute Arterial Stroke; Situations to Evaluate Venous Function; Animal Models and Technologies for Studying Venous Function; 3.3 Proposed Concept of Collateral Homeostasis; Hierarchy of Cerebral Collaterals
- Hemostasis in Both Arterial and Venous Collaterals (Fig. 1.11)Multimodal Monitoring Technologies for Collaterals; 4 Neurovascular Networks as Future Therapeutic Targets; 4.1 Pericytes as a Potential Interventional Target; 4.2 Smooth Muscle Phenotype for Autoregulation; 4.3 Collaterals for Recirculation; 5 Conclusion; References; Chapter 2: Animal Models of Venous Stroke; 1 Selection of Animals for Venous Stroke; 2 Early Cerebral Venous Thrombosis Models; 3 Cerebral Sinus and Venous Thrombosis (CSVT) Model; 3.1 Protocol (for Rat) [5, 14-17]; 3.2 Outcome Evaluation; Physiological Measures
- Local Cerebral Blood Flow (LCBF) MeasurementHistology; 4 Photochemical Dye Induced Cortical Vein Occlusion Model; 4.1 Protocol (for Rat) [20-22, 26]; 4.2 Outcome Evaluation; Local Cerebral Blood Flow Measurement; Histology; 5 Ferric Chloride Induced Superior Sagittal Sinus Thrombosis; 5.1 Protocol; Rat [28, 29]; Mouse [2, 3]; 5.2 Outcome Evaluation; Magnetic Resonance Imaging; Macroscopy, Micro- and Nano-Computed Tomography; References; Chapter 3: Imaging of Cerebral Vein in Acute Brain Injury; 1 Traumatic Brain Injury; 1.1 Computed Tomography (CT); Air in Veins
- 1.2 Susceptibility Weighted Imaging (SWI)Deep Cerebral Veins Asymmetry; Multifocal Signal Loss at Bridging Veins; 1.3 Digital Subtracted Angiography (DSA); Early Venous Filling; 1.4 Summary; 2 Cerebral Venous Thrombosis (CVT); 2.1 Computed Tomography; Dense Triangle Sign and Cord Sign; Focal Distension of the Venous Compartment; 2.2 CT Venography; Empty Delta Sign; 2.3 Magnetic Resonance Imaging; Regular MRI; T1-Weighted Isointense and T2-Weighted Hypointense; Signal Void or Blooming Artifact; High Signal in Sinus; Magnetic Resonance Venography (MRV); Filling Defect in Veins or Sinus
- Dimensions
- unknown
- Extent
- 1 online resource (249 pages).
- File format
- one file format
- Form of item
- online
- Isbn
- 9783319960524
- Level of compression
- unknown
- Media category
- computer
- Media MARC source
- rdamedia
- Media type code
-
- c
- Quality assurance targets
- unknown
- Reformatting quality
- unknown
- Specific material designation
- remote
- System control number
-
- on1076257917
- (OCoLC)1076257917
- Label
- Cerebral venous system in acute and chronic brain injuries, Min Lou, Jianmin Zhang, Yilong Wang, Yan Qu, Wuwei Feng, Xunming Ji, John H. Zhang, editors
- Note
- Hyperintense Signals in Veins or Sinus
- Antecedent source
- file reproduced from an electronic resource
- Carrier category
- online resource
- Carrier category code
-
- cr
- Carrier MARC source
- rdacarrier
- Content category
- text
- Content type code
-
- txt
- Content type MARC source
- rdacontent
- Contents
-
- Intro; Contents; Chapter 1: Neurovascular Network as Future Therapeutic Targets; 1 Introduction; 2 The Mismatch Between Preclinical Models and Clinical Types; 2.1 Recanalization Leads to a Small Infarction; 2.2 Unsuccessful Recanalization Leads to a Small Infarction; 2.3 Recanalization Leads to Bleeding, Edema, and Massive Infarction; 2.4 Vein Compression Involved in Reperfusion Injury; 2.5 Infarction Is Reversible Even After Days; 3 Establishment of New Stroke Pathophysiology to Address Clinical Issues; 3.1 Emphasis on Recirculation: Both Arterial and Venous Blood Flow
- Basic Rules for Recirculation (Fig. 1.7)Anatomy of the Cerebral Venous System; Venous Flow During Ischemic Stroke; Venous Flow During Intracranial Hemorrhage; Venous Flow During Subarachnoid Hemorrhage; Venous Flow During Traumatic Brain Injury; Recirculation as an Emerging Understanding of Stroke and Other Acute Brain Injuries; 3.2 Define Venous Function after Acute Arterial Stroke; Situations to Evaluate Venous Function; Animal Models and Technologies for Studying Venous Function; 3.3 Proposed Concept of Collateral Homeostasis; Hierarchy of Cerebral Collaterals
- Hemostasis in Both Arterial and Venous Collaterals (Fig. 1.11)Multimodal Monitoring Technologies for Collaterals; 4 Neurovascular Networks as Future Therapeutic Targets; 4.1 Pericytes as a Potential Interventional Target; 4.2 Smooth Muscle Phenotype for Autoregulation; 4.3 Collaterals for Recirculation; 5 Conclusion; References; Chapter 2: Animal Models of Venous Stroke; 1 Selection of Animals for Venous Stroke; 2 Early Cerebral Venous Thrombosis Models; 3 Cerebral Sinus and Venous Thrombosis (CSVT) Model; 3.1 Protocol (for Rat) [5, 14-17]; 3.2 Outcome Evaluation; Physiological Measures
- Local Cerebral Blood Flow (LCBF) MeasurementHistology; 4 Photochemical Dye Induced Cortical Vein Occlusion Model; 4.1 Protocol (for Rat) [20-22, 26]; 4.2 Outcome Evaluation; Local Cerebral Blood Flow Measurement; Histology; 5 Ferric Chloride Induced Superior Sagittal Sinus Thrombosis; 5.1 Protocol; Rat [28, 29]; Mouse [2, 3]; 5.2 Outcome Evaluation; Magnetic Resonance Imaging; Macroscopy, Micro- and Nano-Computed Tomography; References; Chapter 3: Imaging of Cerebral Vein in Acute Brain Injury; 1 Traumatic Brain Injury; 1.1 Computed Tomography (CT); Air in Veins
- 1.2 Susceptibility Weighted Imaging (SWI)Deep Cerebral Veins Asymmetry; Multifocal Signal Loss at Bridging Veins; 1.3 Digital Subtracted Angiography (DSA); Early Venous Filling; 1.4 Summary; 2 Cerebral Venous Thrombosis (CVT); 2.1 Computed Tomography; Dense Triangle Sign and Cord Sign; Focal Distension of the Venous Compartment; 2.2 CT Venography; Empty Delta Sign; 2.3 Magnetic Resonance Imaging; Regular MRI; T1-Weighted Isointense and T2-Weighted Hypointense; Signal Void or Blooming Artifact; High Signal in Sinus; Magnetic Resonance Venography (MRV); Filling Defect in Veins or Sinus
- Dimensions
- unknown
- Extent
- 1 online resource (249 pages).
- File format
- one file format
- Form of item
- online
- Isbn
- 9783319960524
- Level of compression
- unknown
- Media category
- computer
- Media MARC source
- rdamedia
- Media type code
-
- c
- Quality assurance targets
- unknown
- Reformatting quality
- unknown
- Specific material designation
- remote
- System control number
-
- on1076257917
- (OCoLC)1076257917
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<div class="citation" vocab="http://schema.org/"><i class="fa fa-external-link-square fa-fw"></i> Data from <span resource="http://link.liverpool.ac.uk/portal/Cerebral-venous-system-in-acute-and-chronic-brain/hGnnnLcsqL8/" typeof="Book http://bibfra.me/vocab/lite/Item"><span property="name http://bibfra.me/vocab/lite/label"><a href="http://link.liverpool.ac.uk/portal/Cerebral-venous-system-in-acute-and-chronic-brain/hGnnnLcsqL8/">Cerebral venous system in acute and chronic brain injuries, Min Lou, Jianmin Zhang, Yilong Wang, Yan Qu, Wuwei Feng, Xunming Ji, John H. Zhang, editors</a></span> - <span property="potentialAction" typeOf="OrganizeAction"><span property="agent" typeof="LibrarySystem http://library.link/vocab/LibrarySystem" resource="http://link.liverpool.ac.uk/"><span property="name http://bibfra.me/vocab/lite/label"><a property="url" href="http://link.liverpool.ac.uk/">Sydney Jones Library, University of Liverpool</a></span></span></span></span></div>
